PEG10 as an oncogene: expression regulatory mechanisms and role in tumor progression-Reference-Cited by-同舟云学术

PEG10 as an oncogene: expression regulatory mechanisms and role in tumor progression

Published:2018-08-13 Issue:1 Volume:18 Page:
ISSN:1475-2867
Container-title:Cancer Cell International
language:en
Short-container-title:Cancer Cell Int

Author:

Xie Tian,Pan Shan,Zheng Hang,Luo Zilv,Tembo Kingsley M.,Jamal Muhammad,Yu Zhongyang,Yu Yao,Xia Jing,Yin Qian,Wang Meng,Yuan Wen,Zhang Qiuping,Xiong Jie^ORCID

Funder

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Oncology

Link

http://link.springer.com/content/pdf/10.1186/s12935-018-0610-3.pdf

Reference76 articles.

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2. Youngson NA, Kocialkowski S, Peel N, Ferguson-Smith AC. A small family of sushi-class retrotransposon-derived genes in mammals and their relation to genomic imprinting. J Mol Evol. 2005;61:481–90.

3. Steplewski A, Krynska B, Tretiakova A, Haas S, Khalili K, Amini S. MyEF-3, a developmentally controlled brain-derived nuclear protein which specifically interacts with myelin basic protein proximal regulatory sequences. Biochem Biophys Res Commun. 1998;243:295–301.

4. Clark MB, Janicke M, Gottesbuhren U, Kleffmann T, Legge M, Poole ES, Tate WP. Mammalian gene PEG10 expresses two reading frames by high efficiency − 1 frameshifting in embryonic-associated tissues. J Biol Chem. 2007;282:37359–69.

5. Ono R, Nakamura K, Inoue K, Naruse M, Usami T, Wakisaka-Saito N, Hino T, Suzuki-Migishima R, Ogonuki N, Miki H, et al. Deletion of Peg10, an imprinted gene acquired from a retrotransposon, causes early embryonic lethality. Nat Genet. 2006;38:101–6.

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