Inflammasomes are important mediators of prostatic inflammation associated with BPH-Reference-Cited by-同舟云学术

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Inflammasomes are important mediators of prostatic inflammation associated with BPH

Published:2015-05-17 Issue:1 Volume:12 Page:
ISSN:1476-9255
Container-title:Journal of Inflammation
language:en
Short-container-title:J Inflamm

Author:

Kashyap Mahendra,Pore Subrata,Wang Zhou,Gingrich Jeffrey,Yoshimura Naoki,Tyagi Pradeep

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry

Link

http://link.springer.com/content/pdf/10.1186/s12950-015-0082-3.pdf

Reference30 articles.

1. Wei JT, Calhoun E, Jacobsen SJ. Urologic diseases in america project: benign prostatic hyperplasia. J Urol. 2008;179(5 Suppl):S75–80. doi:10.1016/j.juro.2008.03.141.

2. Roehrborn CG. BPH progression: concept and key learning from MTOPS, ALTESS, COMBAT, and ALF-ONE. BJU Int. 2008;101 Suppl 3:17–21. doi:10.1111/j.1464-410X.2008.07497.x.

3. St Sauver JL, Jacobson DJ, McGree ME, Lieber MM, Jacobsen SJ. Protective association between nonsteroidal antiinflammatory drug use and measures of benign prostatic hyperplasia. Am J Epidemiol. 2006;164(8):760–8. doi:10.1093/aje/kwj258.

4. Weber K, Schilling JD. Lysosomes integrate metabolic-inflammatory cross-talk in primary macrophage inflammasome activation. J Biol Chem. 2014;289(13):9158–71. doi:10.1074/jbc.M113.531202.

5. Hamakawa T, Sasaki S, Shibata Y, Imura M, Kubota Y, Kojima Y, et al. Interleukin-18 may lead to benign prostatic hyperplasia via thrombospondin-1 production in prostatic smooth muscle cells. Prostate. 2014;74(6):590–601. doi:10.1002/pros.22773.

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