Silencing of SAA1 inhibits palmitate- or high-fat diet induced insulin resistance through suppression of the NF-κB pathway-Reference-Cited by-同舟云学术

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Silencing of SAA1 inhibits palmitate- or high-fat diet induced insulin resistance through suppression of the NF-κB pathway

Published:2019-05-06 Issue:1 Volume:25 Page:
ISSN:1076-1551
Container-title:Molecular Medicine
language:en
Short-container-title:Mol Med

Author:

Wang Yong,Cao Feng,Wang Yang,Yu Gang,Jia Ben-Li

Funder

Doctoral Scientific Research Foundation of the Second Hospital of Anhui Medical University

Natural Science Foundation of Anhui Province: Molecular Mechanism of miR337-3p Regulating HPSE in Gastric Cancer

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1186/s10020-019-0075-4.pdf

Reference57 articles.

1. Ahlin S, Olsson M, Olsson B, Svensson PA, Sjoholm K. No evidence for a role of adipose tissue-derived serum amyloid a in the development of insulin resistance or obesity-related inflammation in hSAA1(+/−) transgenic mice. PLoS One. 2013;8:e72204.

2. Ahlin S, et al. Adipose tissue-derived human serum amyloid a does not affect atherosclerotic lesion area in hSAA1+/−/ApoE−/− mice. PLoS One. 2014;9:e95468.

3. Ajuwon KM, Spurlock ME. Palmitate activates the NF-kappaB transcription factor and induces IL-6 and TNFalpha expression in 3T3-L1 adipocytes. J Nutr. 2005;135:1841–6.

4. Arturi F, et al. Nonalcoholic fatty liver disease is associated with low circulating levels of insulin-like growth factor-I. J Clin Endocrinol Metab. 2011;96:E1640–4.

5. Baranova IN, et al. Serum amyloid A binding to CLA-1 (CD36 and LIMPII analogous-1) mediates serum amyloid A protein-induced activation of ERK1/2 and p38 mitogen-activated protein kinases. J Biol Chem. 2005;280:8031–40.

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