On the primer binding site mutation that appears and disappears during HIV and SIV replication
Author:
Funder
NWO-Chemical Sciences
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Virology
Link
http://link.springer.com/content/pdf/10.1186/s12977-015-0201-5
Reference19 articles.
1. Alexander L, Denekamp L, Czajak S, Desrosiers RC. Suboptimal nucleotides in the infectious, pathogenic simian immunodeficiency virus clone SIVmac239. J Virol. 2001;75(8):4019–22.
2. Fennessey CM, Reid C, Lipkey L, Newman L, Oswald K, Piatak M Jr, Roser JD, Chertova E, Smedley J, Gregory Alvord W, et al. Generation and characterization of a SIVmac239 clone corrected at four suboptimal nucleotides. Retrovirology. 2015;12:49.
3. Soderberg K, Denekamp L, Nikiforow S, Sautter K, Desrosiers RC, Alexander L. A nucleotide substitution in the tRNA(Lys) primer binding site dramatically increases replication of recombinant simian immunodeficiency virus containing a human immunodeficiency virus type 1 reverse transcriptase. J Virol. 2002;76(11):5803–6.
4. Vicenzi E, Dimitrov DS, Engelman A, Migone TS, Purcell DF, Leonard J, Englund G, Martin MA. An integration-defective U5 deletion mutant of human immunodeficiency virus type 1 reverts by eliminating additional long terminal repeat sequences. J Virol. 1994;68(12):7879–90.
5. Das AT, Berkhout B. Efficient extension of a misaligned tRNA-primer during replication of the HIV-1 retrovirus. Nucleic Acids Res. 1995;23(8):1319–26.
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2. Low-level alternative tRNA priming of reverse transcription of HIV-1 and SIV in vivo;Retrovirology;2019-04-04
3. Role of Occult and Post-acute Phase Replication in Protective Immunity Induced with a Novel Live Attenuated SIV Vaccine;PLOS Pathogens;2016-12-21
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