Dihydroisotanshinone I combined with radiation inhibits the migration ability of prostate cancer cells through DNA damage and CCL2 pathway-Reference-Cited by-同舟云学术

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Dihydroisotanshinone I combined with radiation inhibits the migration ability of prostate cancer cells through DNA damage and CCL2 pathway

Published:2018-01-31 Issue:1 Volume:19 Page:
ISSN:2050-6511
Container-title:BMC Pharmacology and Toxicology
language:en
Short-container-title:BMC Pharmacol Toxicol

Author:

Lee I-Yun,Lin Yin-Yin,Yang Yao-Hsu,Lin Yu-Shin,Lin Chun-Liang,Lin Wei-Yu,Cheng Yu-Ching,Shu Li-Hsin,Wu Ching-Yuan

Funder

National Science Council

Chang Gung Memorial Hospital, Linkou

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology

Link

http://link.springer.com/content/pdf/10.1186/s40360-018-0195-4.pdf

Reference45 articles.

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2. Suit HD. Local control and patient survival. Int J Radiat Oncol Biol Phys. 1992;23:653–60.

3. Huen MS, Sy SM, Chen J. BRCA1 and its toolbox for the maintenance of genome integrity. Nat Rev Mol Cell Biol. 2010;11:138–48.

4. Panier S, Durocher D. Regulatory ubiquitylation in response to DNA double-strand breaks. DNA Repair (Amst). 2009;8:436–43.

5. Roos WP, Kaina B. DNA damage-induced cell death: from specific DNA lesions to the DNA damage response and apoptosis. Cancer Lett. 2013;332:237–48.

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