MOK, a pharmacopuncture medicine, regulates thyroid dysfunction in L-thyroxin-induced hyperthyroidism in rats through the regulation of oxidation and the TRPV1 ion channel

Author:

Hwang Ji Hye,Kang Seok Yong,Kang An Na,Jung Hyo Won,Jung Chul,Jeong Jin-Ho,Park Yong-Ki

Funder

Ministry of Health and Welfare

Publisher

Springer Science and Business Media LLC

Subject

Complementary and alternative medicine,General Medicine

Reference46 articles.

1. Zamoner A, Barreto KP, Filho DW, Sell F, Woehl VM, et al. Propylthiouracil-induced congenital hyperthyroidism upregulatesvimentin phosphorylation and depletes antioxidant defenses in immature rat testis. J MolEndocrinol. 2008;40:125–35.

2. Das K, Chainy GB. Modulation of rat liver mitochondrial antioxidant defence system by thyroid hormone. BiochimBiophys Act. 2001;1537:1–13.

3. Subudhi U, Das K, Paital B, Bhanja S, Chainy GB. Alleviation of enhanced oxidative stress and oxygen consumption of L-thyroxine induced hyperthyroid rat liver mitochondria by vitamin E and curcumin. ChemBiolInterac. 2008;173(2):105–14.

4. Chattopadhyay S, Sahoo DK, Subudhi U, Chainy GB. Differential expression profiles of antioxidant enzymes and glutathione redox status in hyperthyroid rats: a temporal analysis. CompBiochemPhysiol C ToxicolPharmacol. 2007;146:383–91.

5. Majima T, Komatsu Y, Doi K, Takagi C, Shigemoto M, et al. Clinical significance of risedronate for osteoporosis in the initial treatment of male patients with Graves' disease. JBone Miner Metab. 2006;24:105–13.

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