Network topology of NaV1.7 mutations in sodium channel-related painful disorders-Reference-Cited by-同舟云学术

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Network topology of NaV1.7 mutations in sodium channel-related painful disorders

Published:2017-02-24 Issue:1 Volume:11 Page:
ISSN:1752-0509
Container-title:BMC Systems Biology
language:en
Short-container-title:BMC Syst Biol

Author:

Kapetis Dimos, ,Sassone Jenny,Yang Yang,Galbardi Barbara,Xenakis Markos N.,Westra Ronald L.,Szklarczyk Radek,Lindsey Patrick,Faber Catharina G.,Gerrits Monique,Merkies Ingemar S. J.,Dib-Hajj Sulayman D.,Mantegazza Massimo,Waxman Stephen G.,Lauria Giuseppe

Funder

Ministero della Salute (IT)

Istitutional funding

European Union 7th Framework Programme

Publisher

Springer Science and Business Media LLC

Subject

Applied Mathematics,Computer Science Applications,Molecular Biology,Modeling and Simulation,Structural Biology

Link

http://link.springer.com/content/pdf/10.1186/s12918-016-0382-0.pdf

Reference85 articles.

1. Catterall WA. Voltage-gated sodium channels at 60: structure, function and pathophysiology. J Physiol. 2012;590:2577–89.

2. Chanda B, Bezanilla F. Tracking voltage-dependent conformational changes in skeletal muscle sodium channel during activation. J Gen Physiol. 2002;120:629–45.

3. Dib-Hajj SD, Yang Y, Black JA, Waxman SG. The Na(V)1.7 sodium channel: from molecule to man. Nat Rev Neurosci. 2013;14:49–62.

4. Cummins TR, Dib-Hajj SD, Waxman SG. Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy. J Neurosci. 2004;24:8232–6.

5. Han C, Rush AM, Dib-Hajj SD, Li S, Xu Z, Wang Y, et al. Sporadic onset of erythermalgia: a gain-of-function mutation in Nav1.7. Ann Neurol. 2006;59:553–8.

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