Differential alterations in the small intestine epithelial cell turnover during acute and chronic infection with Echinostoma caproni (Trematoda)-Reference-Cited by-同舟云学术

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Differential alterations in the small intestine epithelial cell turnover during acute and chronic infection with Echinostoma caproni (Trematoda)

Published:2015-06-18 Issue:1 Volume:8 Page:
ISSN:1756-3305
Container-title:Parasites & Vectors
language:en
Short-container-title:Parasites Vectors

Author:

Cortés Alba,Muñoz-Antoli Carla,Martín-Grau Carla,Esteban J. Guillermo,Grencis Richard K.,Toledo Rafael

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Parasitology

Link

http://link.springer.com/content/pdf/10.1186/s13071-015-0948-5.pdf

Reference39 articles.

1. Maizels RM, Hewitson JP, Smith KA. Susceptibility and immunity to helminth parasites. Curr Opin Immunol. 2012;24:459–66.

2. Grencis RK, Humphreys NE, Bancroft AJ. Immunity to gastrointestinal nematodes: mechanisms and myths. Immunol Rev. 2014;260:183–205.

3. Grencis RK. Immunity to Helminths: Resistance, Regulation, and Susceptibility to Gastrointestinal Nematodes. Annu Rev Immunol. 2015. in press.

4. Da Costa LR. Small-intestinal cell turnover in patients with parasitic infections. Br Med J. 1971;3:281–3.

5. Symons LE. Epithelia cell mitosis and morphology in worm-free regions of the intestines of the rat infected by Nippostrongylusbrasiliensis. J Parasitol. 1978;64:958–9.

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5. FAK Shutdown: Consequences on Epithelial Morphogenesis and Biomarker Expression Involving an Innovative Biomaterial for Tissue Regeneration;International Journal of Molecular Sciences;2021-09-10

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