Microcirculation dysfunction in endotoxic shock rabbits is associated with impaired S-nitrosohemoglobin-mediated nitric oxide release from red blood cells: a preliminary study-Reference-Cited by-同舟云学术

Microcirculation dysfunction in endotoxic shock rabbits is associated with impaired S-nitrosohemoglobin-mediated nitric oxide release from red blood cells: a preliminary study

Published:2019-01-07 Issue:1 Volume:7 Page:
ISSN:2197-425X
Container-title:Intensive Care Medicine Experimental
language:en
Short-container-title:ICMx

Author:

Yao Bo,Liu Da-Wei,Chai Wen-Zhao,Wang Xiao-Ting,Zhang Hong-Min

Publisher

Springer Science and Business Media LLC

Subject

Critical Care and Intensive Care Medicine

Link

http://link.springer.com/content/pdf/10.1186/s40635-018-0215-0.pdf

Reference32 articles.

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2. Ince C (2005) The microcirculation is the motor of sepsis. Crit Care 9(Suppl 4):S13–S19. https://doi.org/10.1186/cc3753.

3. Spronk PE, Zandstra DF, Ince C (2004) Bench-to-bedside review: sepsis is a disease of the microcirculation. Crit Care 8(6):462–468. https://doi.org/10.1186/cc2894.

4. Lam C, Tyml K, Martin C, Sibbald W (1994) Microvascular perfusion is impaired in a rat model of normotensive sepsis. J Clin Invest 94(5):2077–2083. https://doi.org/10.1172/JCI117562

5. Sakr Y, Dubois MJ, De Backer D, Creteur J, Vincent JL (2004) Persistent-microcirculatory alterations are associated with organ failure and death in patients with septic shock. Crit Care Med 32(9):1825–1831

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