Suppression of fibroblast growth factor-2 expression: Possible mechanism underlying methylmercury-induced inhibition of the repair of wounded monolayers of cultured human brain microvascular endothelial cells-Reference-Cited by-同舟云学术

Suppression of fibroblast growth factor-2 expression: Possible mechanism underlying methylmercury-induced inhibition of the repair of wounded monolayers of cultured human brain microvascular endothelial cells

Published:2009 Issue:4 Volume:34 Page:433-439
ISSN:0388-1350
Container-title:The Journal of Toxicological Sciences
language:en
Short-container-title:J. Toxicol. Sci.

Author:

Hirooka Takashi¹,Fujiwara Yasuyuki²,Inoue Shoko³,Shinkai Yasuhiro¹,Yamamoto Chika¹³,Satoh Masahiko²,Yasutake Akira⁴,Eto Komyo⁵,Kaji Toshiyuki¹³

Affiliation:

1. Organization of Frontier Research for Preventive Pharmaceutical Science, Hokuriku University

2. Laboratory of Pharmaceutical Health Sciences, School of Pharmacy, Aichi Gakuin University

3. Department of Environmental Health, Faculty of Pharmaceutical Sciences, Hokuriku University

4. National Institute for Minamata Disease

5. Health and Nursing Facilities for the Aged, Jushindai, Shinwakai

Publisher

Japanese Society of Toxicology

Subject

Toxicology

Link

http://www.jstage.jst.go.jp/article/jts/34/4/34_4_433/_pdf

Reference25 articles.

1. Abbott, J.N., Rönnbäck, L. and Hansson, E. (2006): Astrocyte-endothelial interactions at the blood-brain barrier. Nat. Rev. Neurosci., 7, 41-53.

2. Perlecan, basal lamina proteoglycan, promotes basic fibroblast growth factor-receptor binding, mitogenesis, and angiogenesis

3. Differential structural requirements of heparin and heparan sulfate proteoglycans that promote binding of basic fibroblast growth factor to its receptor.

4. Methylmercury Poisoning in Iraq

5. Effects of Methylmercury on the Microvasculature of the Developing Brain

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