Analysis of microRNA expression profiling during paraquat-induced injury of murine lung alveolar epithelial cells
Author:
Affiliation:
1. Department of Emergency, The First Affiliated Hospital of China Medical University, China
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/45/8/45_423/_pdf
Reference45 articles.
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2. Bartel, D.P. (2004): MicroRNAs: genomics, biogenesis, mechanism, and function. Cell, 116, 281-297.
3. Chen, F., Qi, S., Zhang, X., Wu, J., Yang, X. and Wang, R. (2019a): miR-23a-3p suppresses cell proliferation in oral squamous cell carcinomas by targeting FGF2 and correlates with a better prognosis: miR-23a-3p inhibits OSCC growth by targeting FGF2. Pathol. Res. Pract., 215, 660-667.
4. Chen, W., Zhou, Z.Q., Ren, Y.Q., Zhang, L., Sun, L.N., Man, Y.L. and Wang, Z.K. (2019b): Effects of long non-coding RNA LINC00667 on renal tubular epithelial cell proliferation, apoptosis and renal fibrosis via the miR-19b-3p/LINC00667/CTGF signaling pathway in chronic renal failure. Cell. Signal., 54, 102-114.
5. Cui, S., Nian, Q., Chen, G., Wang, X., Zhang, J., Qiu, J. and Zhang, Z. (2019): Ghrelin ameliorates A549 cell apoptosis caused by paraquat via p38-MAPK regulated mitochondrial apoptotic pathway. Toxicology, 426, 152267.
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