Effects of continuous thiamine intake on onset and progression of type 2 diabetes in leptin-receptor deficient mice
Author:
Affiliation:
1. Department of Pharmacotherapeutics, Faculty of Pharmacy, Osaka Medical and Pharmaceutical University
Publisher
Japanese Society of Toxicology
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/fts/10/2/10_83/_pdf
Reference44 articles.
1. Abboud, M.R., Alexander, D. and Najjar, S.S. (1985): Diabetes mellitus, thiamine-dependent megaloblastic anemia, and sensorineural deafness associated with deficient alpha-ketoglutarate dehydrogenase activity. J. Pediatr., 107, 537-541.
2. Afshin, A., Forouzanfar, M.H., Reitsma, M.B., Sur, P., Estep, K., et al. and GBD 2015 Obesity Collaborators. (2017): Health Effects of Overweight and Obesity in 195 Countries over 25 Years. N. Engl. J. Med., 377, 13-27.
3. Alzahrani, A.S., Baitei, E., Zou, M. and Shi, Y. (2006): Thiamine transporter mutation: an example of monogenic diabetes mellitus. Eur. J. Endocrinol., 155, 787-792.
4. Babaei-Jadidi, R., Karachalias, N., Ahmed, N., Battah, S. and Thornalley, P.J. (2003): Prevention of incipient diabetic nephropathy by high-dose thiamine and benfotiamine. Diabetes, 52, 2110-2120.
5. Babaei-Jadidi, R., Karachalias, N., Kupich, C., Ahmed, N. and Thornalley, P.J. (2004): High-dose thiamine therapy counters dyslipidaemia in streptozotocin-induced diabetic rats. Diabetologia, 47, 2235-2246.
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