Antidiabetic agent did not impair spermatogenesis in spontaneously hyperglycemic and diabetic rats
Author:
Affiliation:
1. Graduate School of Veterinary Science, Azabu University
2. TEIJIN PHARMA LIMITED
3. TEIJIN LIMITED
Publisher
Japanese Society of Toxicology
Link
https://www.jstage.jst.go.jp/article/fts/8/4/8_97/_pdf
Reference14 articles.
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2. Bonds, D.E., Miller, M.E., Bergenstal, R.M., Buse, J.B., Byington, R.P., Cutler, J.A., Dudl, R.J., Ismail-Beigi, F., Kimel, A.R., Hoogwerf, B., Horowitz, K.R., Savage, P.J., Seaquist, E.R., Simmons, D.L., Sivitz, W.I., Speril-Hillen, J.M. and Sweeney, M.E. (2010): The association between symptomatic, severe hypoglycaemia and mortality in type 2 diabetes: retrospective epidemiological analysis of the ACCORD study. BMJ, 340, b4909.
3. Ferre, T., Riu, E., Bosch, F. and Valera, A. (1996): Evidence from transgenic mice that glucokinase is rate limiting for glucose utilization in the liver. FASEB J., 10, 1213-1218.
4. International Hypoglycaemia Study Group. (2017): Glucose concentrations of less than 3.0 mmol/l (54 mg/dl) should be reported in clinical trials: a joint position statement of the American Diabetes Association and the European Association for the Study of Diabetes. Diabetes Care, 40, 155-157.
5. King, A.J. (2002): The use of animal models in diabetes research. Br. J. Pharmacol., 166, 877-894.
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