Enhanced proliferative response of hepatocytes to combined inhalation and oral exposures to N,N-dimethylformamide in male rats

Author:

Ohbayashi Hisao1,Yamazaki Kazunori1,Aiso Shigetoshi1,Nagano Kasuke1,Fukushima Shoji1,Ohta Hisayoshi2

Affiliation:

1. Japan Bioassay Research Center, Japan Industrial Safety and Health Association

2. School of Allied Health Sciences, Kitasato University

Publisher

Japanese Society of Toxicology

Subject

Toxicology

Reference45 articles.

1. Butterworth, B.E., Popp, J.A., Conolly, R.B. and Goldsworthy, T.L. (1992): Chemically induced cell proliferation in carcinogenesis. In Mechanisms of Carcinogenesis in Risk Identification (Vainio, H., Magee, P.N., McGregor, D.B. and McMichael A.J., eds.), IARC Scientific Publication, Number 116, pp. 279-305, IARC, Lyon.

2. Hepatotoxicity and P-4502E1-dependent metabolic oxidation of N,N-dimethylformamide in rats and mice

3. Subchronic Inhalation Toxicity of Dimethylformamide in Rats and Mice

4. Liver function alterations in synthetic leather workers exposed to dimethylformamide

5. Fleming, L.E., Shalat, S.L. and Redlich, C.A. (1990): Liver injury in workers exposed to dimethylformamide. Scan. J. Work Environ. Health, 16, 289-292.

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