Involvement of epithelial-mesenchymal transition in methotrexate-induced pulmonary fibrosis
Author:
Affiliation:
1. Division of Clinical Pharmacy, Department of Pharmacotherapeutics, School of Pharmacy, Showa University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/39/2/39_319/_pdf
Reference40 articles.
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2. Bremnes, R.M., Veve, R., Gabrielson, E., Hirsch, F.R., Baron, A., Bemis, L., Gemmill, R.M., Drabkin, H.A. and Franklin, W.A. (2002): High-throughput tissue microarray analysis used to evaluate biology and prognostic significance of the E-cadherin pathway in non-small-cell lung cancer. J. Clin. Oncol., 20, 2417-2428.
3. Bucala, R., Spiegel, L.A., Chesney, J., Hogan, M. and Cerami, A. (1994): Circulating fibrocytes define a new leukocyte subpopulation that mediates tissue repair. Mol. Med., 1, 71-81.
4. Carson, C.W., Cannon, G.W., Egger, M.J., Ward, J.R. and Clegg, D.O. (1987): Pulmonary disease during the treatment of rheumatoid arthritis with low dose pulse methotrexate. Semin. Arthritis Rheum., 16, 186-195.
5. Chen, X.F., Zhang, H.J., Wang, H.B., Zhu, J., Zhou, W.Y., Zhang, H., Zhao, M.C., Su, J.M., Gao, W., Zhang, L., Fei, K., Zhang, H.T. and Wang, H.Y. (2012): Transforming growth factor-β1 induces epithelial-to-mesenchymal transition in human lung cancer cells via PI3K/Akt and MEK/Erk1/2 signaling pathways. Mol. Biol. Rep., 39, 3549-3556.
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