Differential toxicity between perfluorooctane sulfonate (PFOS) and perfluorooctanoic acid (PFOA)
Author:
Affiliation:
1. Iwate Institute of Environmental Health Sciences
2. Nippon Veterinary and Life Science University
3. Iwate University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/41/Special/41_SP27/_pdf
Reference57 articles.
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2. Beggs, K.M., McGreal, S.R., McCarthy, A., Gunewardena, S., Lampe, J.N., Lau, C. and Apte, U. (2016): The role of hepatocyte nuclear factor 4-alpha in perfluorooctanoic acid- and perfluorooctanesulfonic acid-induced hepatocellular dysfunction. Toxicol. Appl. Pharmacol., 304, 18-29.
3. Butenhoff, J.L., Ehresman, D.J., Chang, S.-C., Parker, G.A. and Stump, D.G. (2009): Gestational and lactational exposure to potassium perfluorooctanesulfonate (K+PFOS) in rats: Developmental neurotoxicity. Reprod. Toxicol., 27, 319-330.
4. Buhrke, T., Krüger, E., Pevny, S., Rößler, M., Bitter, K. and Lampen, A. (2015): Perfluorooctanoic acid (PFOA) affects distinct molecular signalling pathways in human primary hepatocytes. Toxicology, 333, 53-62.
5. Elcombe, C.R., Elcombe, B.M., Foster, J.R., Farrar, D.G., Jung, R., Chang, S.C., Kennedy, G.L. and Butenhoff, J.L. (2010): Hepatocellular hypertrophy and cell proliferation in Sprague-Dawley rats following dietary exposure to ammonium perfluorooctanoate occurs through increased activation of the xenosensor nuclear receptors PPARα and CAR/PXR. Arch. Toxicol., 84, 787-798.
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