MaxiK channel roles in blood vessel relaxations induced by endothelium-derived relaxing factors and their molecular mechanisms-Reference-Cited by-同舟云学术

MaxiK channel roles in blood vessel relaxations induced by endothelium-derived relaxing factors and their molecular mechanisms

Published:2004 Issue:4,5 Volume:40 Page:125-153
ISSN:0916-8737
Container-title:Journal of Smooth Muscle Research
language:en
Short-container-title:J. Smooth Muscle Res.

Author:

Tanaka Yoshio¹,Koike Katsuo¹,Toro Ligia²³⁴

Affiliation:

1. Department of Chemical Pharmacology, Toho University School of Pharmaceutical Sciences

2. Department of Anesthesiology, Division of Molecular Medicine, University of California

3. Department of Molecular and Medical Pharmacology, University of California

4. Brain Research Institute, David Geffen School of Medicine, University of California

Publisher

Japan Society of Smooth Muscle Research

Subject

General Medicine,Physiology

Link

https://www.jstage.jst.go.jp/article/jsmr/40/4,5/40_4,5_125/_pdf

Reference152 articles.

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2. Akiba, T., Miyazaki, M. and Toda, N. (1986). Vasodilator actions of TRK-100, a new prostaglandin I₂ analogue. Br. J. Pharmacol. 89: 703-711.

3. Alioua, A., Huggins, J.P. and Rousseau, E. (1995). PKG-Iα phosphorylates the α-subunit and upregulates reconstituted GK_Ca channels from tracheal smooth muscle. Am. J. Physiol. 268: L1057-L1063.

4. Alioua, A., Tanaka, Y., Wallner, M., Hofmann, P., Ruth, P., Meera, P. and Toro, L. (1998). The large conductance, voltage-dependent and calcium-sensitive K⁺ channel, hslo, is a target of cGMP-dependent protein kinase phosphorylation in vivo. J. Biol. Chem. 273: 32950-32956.

5. Alioua, A., Mahajan, A., Nishimura, K., Zarei, M.M., Stefani, E. and Toro, L. (2002). Coupling of c-Src to large conductance voltage- and Ca²⁺-activated K⁺ channels as a new mechanism of agonist-induced vasoconstriction. Proc. Natl. Acad. Sci. U.S.A. 99: 14560-14565.

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