Germ-Free Condition and the Susceptibility of Balb/C Mice to Post-Thymectomy Autoimmune Gastritis
Author:
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
http://www.tandfonline.com/doi/pdf/10.3109/08916939209146132
Reference7 articles.
1. Molecular mimicry and autoimmune disease
2. The 60- to 90-kDa parietal cell autoantigen associated with autoimmune gastritis is a beta subunit of the gastric H+/K(+)-ATPase (proton pump).
3. Genetic studies on experimental autoimmune gastritis induced by neonatal thymectomy using recombinant inbred strains between a high-incidence strain, BALB/c, and a low-incidence strain, DBA/2
4. T-Cell Responses to Mls and to Bacterial Proteins that Mimic its Behavior#
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1. Induction of autoimmune gastritis by neonatal thymectomy requires autoantibodies and is prevented by anti-FcγR antibodies;Cellular Immunology;2016-02
2. T Lymphocyte-Mediated Control of Autoimmunity;Novartis Foundation Symposia;2007-09-28
3. Genetic Control of Susceptibility to Autoimmune Gastritis;International Reviews of Immunology;2005-01
4. CD4+CD45RBHi Interleukin-4 Defective T Cells Elicit Antral Gastritis and Duodenitis;The American Journal of Pathology;2004-10
5. Interactions between commensal intestinal bacteria and the immune system;Nature Reviews Immunology;2004-06
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