The role of cell death in the pathogenesis of autoimmune disease: HMGB1 and microparticles as intercellular mediators of inflammation-Reference-Cited by-同舟云学术

The role of cell death in the pathogenesis of autoimmune disease: HMGB1 and microparticles as intercellular mediators of inflammation

Published:2008-08 Issue:4 Volume:18 Page:319-326
ISSN:1439-7595
Container-title:Modern Rheumatology
language:en
Short-container-title:Modern Rheumatology

Author:

Ardoin Stacy P.¹,Pisetsky David S.²

Affiliation:

1. Departments of Internal Medicine and Pediatrics, Divisions of Rheumatology and Pediatric Rheumatology, Duke University Medical Center, DUMC Box 3212, Durham, NC 27710, USA

2. Department of Internal Medicine, Division of Rheumatology and Medical Research Service, Durham VA Hospital, Durham, NC, USA

Publisher

Oxford University Press (OUP)

Subject

Rheumatology

Link

https://www.tandfonline.com/doi/pdf/10.3109/s10165-008-0054-z

Reference69 articles.

1. A human placental cDNA clone that encodes nonhistone chromosomal protein HMG-1

2. The DNA binding site of HMG1 protein is composed of two similar segments (HMG boxes), both of which have counterparts in other eukaryotic regulatory proteins.

3. High Mobility Group 1 Protein Is Not Stably Associated with the Chromosomes of Somatic Cells

4. High mobility group protein-1 (HMG-1) is a unique activator of p53

5. HMGB1 interacts differentially with members of the Rel family of transcription factors

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