Interaction of an α+-Thalassemia Deletion with Either a Highly Unstable α-Globin Variant (α2, Codon 59, GGC→GAC) or a Nondeletional α-Thalassemia Mutation (AATAA→AATAAG): Comparison of Phenotypes Illustrating “Dominant” α-Thalassemia-Reference-Cited by-同舟云学术

Interaction of an α+-Thalassemia Deletion with Either a Highly Unstable α-Globin Variant (α2, Codon 59, GGC→GAC) or a Nondeletional α-Thalassemia Mutation (AATAA→AATAAG): Comparison of Phenotypes Illustrating “Dominant” α-Thalassemia

Published:1999-01 Issue:4 Volume:23 Page:325-337
ISSN:0363-0269
Container-title:Hemoglobin
language:en
Short-container-title:Hemoglobin

Author:

Traeger-Synodinos J.,Metaxotou-Mavrommati A.,Karagiorga M.,Vrettou C.,Papassotiriou I.,Stamoulakatou A.,Kanavakis E.

Publisher

Informa UK Limited

Subject

Biochemistry, medical,Clinical Biochemistry,Genetics(clinical),Hematology

Link

http://www.tandfonline.com/doi/pdf/10.3109/03630269909090748

Reference30 articles.

1. DOMINANT Î² THALASSAEMIA: MOLECULAR BASIS AND PATHOPHYSIOLOGY

2. Molecular basis for dominantly inherited inclusion body beta-thalassemia.

3. The Thalassemia Repository (Ninth Edition; Part II)

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