Impaired Breakdown of Bradykinin and Its Metabolites as a Possible Cause for Pulmonary Edema in COVID-19 Infection-Reference-Cited by-同舟云学术

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Impaired Breakdown of Bradykinin and Its Metabolites as a Possible Cause for Pulmonary Edema in COVID-19 Infection

Published:2020-06-11 Issue:07 Volume:46 Page:835-837
ISSN:0094-6176
Container-title:Seminars in Thrombosis and Hemostasis
language:en
Short-container-title:Semin Thromb Hemost

Author:

de Maat Steven¹,de Mast Quirijn²,Danser A.H. Jan³,van de Veerdonk Frank L.²,Maas Coen¹

Affiliation:

1. CDL Research, University Medical Center Utrecht, Utrecht University, Utrecht, The Netherlands

2. Department of Internal Medicine, Radboudumc Center for Infectious Diseases, Radboud University Medical Center, Nijmegen, The Netherlands

3. Department of Internal Medicine, Division of Pharmacology and Vascular Medicine, Erasmus MC, Rotterdam, The Netherlands

Publisher

Georg Thieme Verlag KG

Subject

Cardiology and Cardiovascular Medicine,Hematology

Link

http://www.thieme-connect.de/products/ejournals/pdf/10.1055/s-0040-1712960.pdf

Reference27 articles.

1. Membrane ectopeptidases targeted by human coronaviruses;B J Bosch;Curr Opin Virol,2014

2. A human homolog of angiotensin-converting enzyme. Cloning and functional expression as a captopril-insensitive carboxypeptidase;S R Tipnis;J Biol Chem,2000

3. A pneumonia outbreak associated with a new coronavirus of probable bat origin;P Zhou;Nature,2020

4. Tissue distribution of ACE2 protein, the functional receptor for SARS coronavirus. A first step in understanding SARS pathogenesis;I Hamming;J Pathol,2004

5. Angiotensin-converting enzyme 2 protects from severe acute lung failure;Y Imai;Nature,2005

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