Glucose desensitization in INS-1 cells: Evidence of impaired function caused by glucose metabolite(s) rather than by the glucose molecule per se
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Cited by 11 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. KATP Channels and the Metabolic Regulation of Insulin Secretion in Health and Disease: The 2022 Banting Medal for Scientific Achievement Award Lecture;Diabetes;2023-05-19
2. Glucokinase activity in diabetes: too much of a good thing?;Trends in Endocrinology & Metabolism;2023-02
3. Acclimation of C2C12 myoblasts to physiological glucose concentrations for in vitro diabetes research;Life Sciences;2018-10
4. Disrupted dynamics of F-actin and insulin granule fusion in INS-1 832/13 beta-cells exposed to glucotoxicity: partial restoration by glucagon-like peptide 1;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2016-08
5. Glucokinase mediates coupling of glycolysis to mitochondrial metabolism but not to beta cell damage at high glucose exposure levels;Diabetologia;2011-04-12
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