Wild-Type Human PRSS2 and PRSS1R122H Cooperatively Initiate Spontaneous Hereditary Pancreatitis in Transgenic Mice
Author:
Funder
Mayo Clinic
NIH
U.S. Department of Defense
Publisher
Elsevier BV
Subject
Gastroenterology,Hepatology
Reference10 articles.
1. Early Intra-Acinar Events in Pathogenesis of Pancreatitis
2. Intracellular activation of trypsinogen in transgenic mice induces acute but not chronic pancreatitis
3. A preclinical model of chronic pancreatitis driven by trypsinogen autoactivation
4. Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene
5. Increased Activation of Hereditary Pancreatitis-associated Human Cationic Trypsinogen Mutants in Presence of Chymotrypsin C
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