The atypical receptor CCRL2 is required for CXCR2-dependent neutrophil recruitment and tissue damage-Reference-Cited by-同舟云学术

The atypical receptor CCRL2 is required for CXCR2-dependent neutrophil recruitment and tissue damage

Published:2017-09-07 Issue:10 Volume:130 Page:1223-1234
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Del Prete Annalisa¹²^ORCID,Martínez-Muñoz Laura³,Mazzon Cristina²,Toffali Lara⁴,Sozio Francesca¹²,Za Lorena⁵,Bosisio Daniela¹,Gazzurelli Luisa¹,Salvi Valentina¹^ORCID,Tiberio Laura¹^ORCID,Liberati Chiara⁵,Scanziani Eugenio⁶^ORCID,Vecchi Annunciata²,Laudanna Carlo⁴,Mellado Mario³,Mantovani Alberto²⁷⁸^ORCID,Sozzani Silvano¹²^ORCID

Affiliation:

1. Department of Molecular and Translational Medicine, University of Brescia, Brescia, Italy;

2. Humanitas Clinical and Research Center, Rozzano-Milano, Italy;

3. Department of Immunology and Oncology, Centro Nacional de Biotecnología, Madrid, Spain;

4. Department of Medicine, University of Verona, Verona, Italy;

5. Axxam Discovery Biology, Bresso, Italy;

6. Department of Veterinary Sciences and Public Health, University of Milan, Milan, Italy;

7. Laboratory of Immunology, Humanitas University, Rozzano-Milano, Italy; and

8. The William Harvey Research Institute, Queen Mary University of London, London, United Kingdom

Abstract

Key Points CCRL2 is required for CXCR2-dependent neutrophil recruitment. CCRL2 forms heterodimers with CXCR2 and regulates CXCR2 signaling.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/130/10/1223/1402090/blood777680.pdf

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