Cytoplasmic localization of phosphorylated STAT5 in human acute myeloid leukemia is inversely correlated with Flt3-ITD
Author:
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/110/7/2775/483786/zh801907002775.pdf
Reference6 articles.
1. Flow cytometric measurement of phosphorylated STAT5 in AML: lack of specific association with FLT3 internal tandem duplications.;Pallis;Leuk Res,2003
2. Constitutive activation of Stat5 promotes its cytoplasmic localization and association with PI 3-kinase in myeloid leukemias.;Harir;Blood,2007
3. Bone marrow phospho-STAT5 expression in non-CML chronic myeloproliferative disorders correlates with JAK2 V617F mutation and provides evidence of in vivo JAK2 activation.;Aboudola;Am J Surg Pathol,2007
4. Signal transducer and activator of transcription-5 activation and breast cancer prognosis.;Nevalainen;J Clin Oncol,2004
5. Rac1 and a GTPase-activating protein, MgcRacGAP, are required for nuclear translocation of STAT transcription factors.;Kawashima;J Cell Biol,2006
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2. STAT5 is Expressed in CD34+/CD38− Stem Cells and Serves as a Potential Molecular Target in Ph-Negative Myeloproliferative Neoplasms;Cancers;2020-04-21
3. pSTAT3/pSTAT5 Signaling Patterns in Molecularly Defined Subsets of Myeloproliferative Neoplasms;Applied Immunohistochemistry & Molecular Morphology;2018-02
4. A rapid method for quantifying cytoplasmic versus nuclear localization in endogenous peripheral blood leukocytes by conventional flow cytometry;Cytometry Part A;2017-03-30
5. STAT5 in hematopoietic stem cell biology and transplantation;JAK-STAT;2013-10-16
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