C-Terminal Cleavage of the Amyloid-β Protein Precursor at Asp664: A Switch Associated with Alzheimer's Disease-Reference-Cited by-同舟云学术

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C-Terminal Cleavage of the Amyloid-β Protein Precursor at Asp664: A Switch Associated with Alzheimer's Disease

Published:2008-02-25 Issue:1 Volume:13 Page:1-16
ISSN:1875-8908
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Banwait Surita¹,Galvan Veronica¹,Zhang Junli¹,Gorostiza Olivia F.¹,Ataie Marina¹,Huang Wei¹,Crippen Danielle¹,Koo Edward H.²,Bredesen Dale E.¹³

Affiliation:

1. Buck Institute for Age Research, 8001 Redwood Blvd., Novato, CA 94945, USA

2. University of California, San Diego, Department of Neuroscience, La Jolla, CA 92093, USA

3. University of California, San Francisco, Department of Neurology, San Francisco, CA, 94143, USA

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Cited by 23 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Amyloidogenesis: What Do We Know So Far?;International Journal of Molecular Sciences;2022-11-12

2. Cell Death Related Proteins Beyond Apoptosis in the CNS;Frontiers in Cell and Developmental Biology;2022-01-14

3. Neuregulin 1/ErbB4/Akt signaling attenuates cytotoxicity mediated by the APP-CT31 fragment of amyloid precursor protein;Experimental and Molecular Pathology;2021-06

4. Caspase Activation and Caspase-Mediated Cleavage of APP Is Associated with Amyloid β-Protein-Induced Synapse Loss in Alzheimer’s Disease;Cell Reports;2020-06

5. The mitochondria-targeted antioxidant MitoQ inhibits memory loss, neuropathology, and extends lifespan in aged 3xTg-AD mice;Molecular and Cellular Neuroscience;2019-12

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