An 18-mer Peptide Derived from Prosaposin Ameliorates the Effects of Aβ1–42 Neurotoxicity on Hippocampal Neurogenesis and Memory Deficit in Mice-Reference-Cited by-同舟云学术

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An 18-mer Peptide Derived from Prosaposin Ameliorates the Effects of Aβ1–42 Neurotoxicity on Hippocampal Neurogenesis and Memory Deficit in Mice

Published:2016-08-03 Issue:3 Volume:53 Page:1173-1192
ISSN:1387-2877
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Gao Hui-ling¹,Li Cheng²,Nabeka Hiroaki³,Shimokawa Tetsuya³,Wang Zhan-You¹,Cao Ya-ming²,Matsuda Seiji³

Affiliation:

1. College of Life and Health Sciences, Northeastern University, Shenyang, China

2. Department of Immunology, China Medical University, Shenyang, China

3. Department of Anatomy and Embryology, Ehime University Graduate School of Medicine, Toon, Ehime, Japan

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Reference86 articles.

1. Identification of prosaposin as a neurotrophic factor;O’Brien;Proc Natl Acad Sci U S A,1994

2. Identification of the neurotrophic factor sequence of prosaposin;O’Brien;FASEB J,1995

3. Soluble amyloid beta-protein dimers isolated from Alzheimer cortex directly induce tau hyperphosphorylation and neuritic degeneration;Jin;Proc Natl Acad Sci U S A,2011

4. Distribution of prosaposin in the rat nervous system;Hosoda;Cell Tissue Res,2007

5. Distribution of prosaposin-like immunoreactivity in rat brain;Kondoh;J Comp Neurol,1993

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