High Dietary Advanced Glycation End Products Impair Mitochondrial and Cognitive Function-Reference-Cited by-同舟云学术

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High Dietary Advanced Glycation End Products Impair Mitochondrial and Cognitive Function

Published:2020-06-30 Issue:1 Volume:76 Page:165-178
ISSN:1387-2877
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Akhter Firoz¹²,Chen Doris¹,Akhter Asma¹²,Sosunov Alexander A.³,Chen Allen¹,McKhann Guy M.³,Yan Shi Fang¹³,Yan Shirley ShiDu¹²

Affiliation:

1. Department of Pharmacology and Toxicology and Higuchi Biosciences Center, University of Kansas, Lawrence, KS, USA

2. Department of Surgery, Columbia University, New York, NY, USA

3. Department of Neurological Surgery and Surgery, Columbia University, New York, NY, USA

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Reference50 articles.

1. Enhanced cellular oxidant stress by the interaction of advanced glycation end products with their receptors/binding proteins;Yan;J Biol Chem,1994

2. Genetic deficiency of neuronal RAGE protects against AGE-induced synaptic injury;Zhang;Cell Death Dis,2014

3. Formation of glyoxal, methylglyoxal and 3-deoxyglucosone in the glycation of proteins by glucose;Thornalley;Biochem J,1999

4. Protein and nucleotide damage by glyoxal and methylglyoxal in physiological systems–role in ageing and disease;Thornalley;Drug Metabol Drug Interact,2008

5. Pathways of the Maillard reaction under physiological conditions;Henning;Glycoconj J,2016

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