Altered zinc balance in the Atp7b−/− mouse reveals a mechanism of copper toxicity in Wilson disease-Reference-Cited by-同舟云学术

Altered zinc balance in the Atp7b−/− mouse reveals a mechanism of copper toxicity in Wilson disease

Published:2018 Issue:11 Volume:10 Page:1595-1606
ISSN:1756-5901
Container-title:Metallomics
language:en
Short-container-title:Metallomics

Author:

Meacham Kelsey A.¹²³⁴^ORCID,Cortés María Paz⁵⁶⁷⁸⁹^ORCID,Wiggins Eve M.¹²³⁴^ORCID,Maass Alejandro⁵⁶⁷⁸⁹,Latorre Mauricio⁵⁶⁷⁸⁹,Ralle Martina¹⁰¹¹¹²⁴,Burkhead Jason L.¹²³⁴^ORCID

Affiliation:

1. Department of Biological Sciences

2. University of Alaska Anchorage

3. Anchorage

4. USA

5. Mathomics

6. Universidad de Chile

7. Santiago

8. Chile

9. Center of Mathematical Modeling (UMI2807 UCHILE-CNRS)

10. Molecular & Medical Genetics Department

11. Oregon Health & Science University School of Medicine

12. Portland

Abstract

Copper accumulation in the Atp7b^−/− model of Wilson disease impacts zinc distribution.

Funder

National Institute of General Medical Sciences

National Center for Research Resources

National Institute of Diabetes and Digestive and Kidney Diseases

Fondo Nacional de Desarrollo Científico y Tecnológico

National Institute of Dental and Craniofacial Research

Publisher

Oxford University Press (OUP)

Subject

Metals and Alloys,Biochemistry,Biomaterials,Biophysics,Chemistry (miscellaneous)

Link

http://pubs.rsc.org/en/content/articlepdf/2018/MT/C8MT00199E

Reference67 articles.

1. Evolving Perspectives in Wilson Disease: Diagnosis, Treatment and Monitoring

2. The Wilson disease gene is a putative copper transporting P–type ATPase similar to the Menkes gene

3. Cellular Copper Transport and Metabolism

4. Wilson Disease: Pathogenesis and Clinical Considerations in Diagnosis and Treatment

5. Copper in disorders with neurological symptoms: Alzheimer’s, Menkes, and Wilson diseases

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