Oligodendroglia metabolically support axons and contribute to neurodegeneration-Reference-Cited by-同舟云学术

Oligodendroglia metabolically support axons and contribute to neurodegeneration

Published:2012-07 Issue:7408 Volume:487 Page:443-448
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Lee Youngjin,Morrison Brett M.,Li Yun,Lengacher Sylvain,Farah Mohamed H.,Hoffman Paul N.,Liu Yiting,Tsingalia Akivaga,Jin Lin,Zhang Ping-Wu,Pellerin Luc,Magistretti Pierre J.,Rothstein Jeffrey D.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature11314.pdf

Reference49 articles.

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2. Garbern, J. Y. et al. Patients lacking the major CNS myelin protein, proteolipid protein 1, develop length-dependent axonal degeneration in the absence of demyelination and inflammation. Brain 125, 551–561 (2002)

3. Griffiths, I. et al. Axonal swellings and degeneration in mice lacking the major proteolipid of myelin. Science 280, 1610–1613 (1998)

4. Lappe-Siefke, C. et al. Disruption of Cnp1 uncouples oligodendroglial functions in axonal support and myelination. Nature Genet. 33, 366–374 (2003)

5. Fünfschilling, U. et al. Glycolytic oligodendrocytes maintain myelin and long-term axonal integrity. Nature 485, 517–521 (2012)

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