NR2F1 controls tumour cell dormancy via SOX9- and RARβ-driven quiescence programmes-Reference-Cited by-同舟云学术

NR2F1 controls tumour cell dormancy via SOX9- and RARβ-driven quiescence programmes

Published:2015-01-30 Issue:1 Volume:6 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Sosa Maria Soledad,Parikh Falguni,Maia Alexandre Gaspar,Estrada Yeriel,Bosch Almudena,Bragado Paloma,Ekpin Esther,George Ajish,Zheng Yang,Lam Hung-Ming,Morrissey Colm,Chung Chi-Yeh,Farias Eduardo F.,Bernstein Emily,Aguirre-Ghiso Julio A.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms7170.pdf

Reference48 articles.

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2. Sosa, M. S., Bragado, P. & Aguirre-Ghiso, J. A. Mechanisms of disseminated cancer cell dormancy: an awakening field. Nat. Rev. Cancer 14, 611–622 (2014).

3. Adam, A. P. et al. Computational identification of a p38SAPK-regulated transcription factor network required for tumor cell quiescence. Cancer Res. 69, 5664–5672 (2009).

4. Onder, T. T. et al. Chromatin-modifying enzymes as modulators of reprogramming. Nature 483, 598–602 (2012).

5. Marion, R. M. et al. A p53-mediated DNA damage response limits reprogramming to ensure iPS cell genomic integrity. Nature 460, 1149–1153 (2009).

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