A new pathway that regulates 53BP1 stability implicates Cathepsin L and vitamin D in DNA repair-Reference-Cited by-同舟云学术

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A new pathway that regulates 53BP1 stability implicates Cathepsin L and vitamin D in DNA repair

Published:2011-07-12 Issue:16 Volume:30 Page:3383-3396
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:

Author:

Gonzalez-Suarez Ignacio¹,Redwood Abena B¹,Grotsky David A¹,Neumann Martin A¹,Cheng Emily H-Y²,Stewart Colin L³,Dusso Adriana¹⁴,Gonzalo Susana¹

Affiliation:

1. Radiation and Cancer Biology Division, Department of Radiation Oncology, Washington University School of Medicine; St Louis MO USA

2. Human Oncology and Pathogenesis Program, Memorial Sloan-Kettering Cancer Center; New York NY USA

3. Institute of Medical Biology, Immunos Singapore; Singapore

4. Division of Experimental Nephrology, IRB Lleida; Spain

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference49 articles.

1. Inactivation of the lamin A/C gene by CpG island promoter hypermethylation in hematologic malignancies, and its association with poor survival in nodal diffuse large B-cell lymphoma;Agrelo;J Clin Oncol,2005

2. Cystatin D is a candidate tumor suppressor gene induced by vitamin D in human colon cancer cells;Alvarez-Diaz;J Clin Invest,2009

3. Structural basis for the methylation state-specific recognition of histone H4-K20 by 53BP1 and Crb2 in DNA repair;Botuyan;Cell,2006

4. 53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers;Bouwman;Nat Struct Mol Biol,2010

5. Nuclear A-type lamins are differentially expressed in human lung cancer subtypes;Broers;Am J Pathol,1993

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