DNA damage: a trigger of innate immunity but a requirement for adaptive immune homeostasis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Energy Engineering and Power Technology,Fuel Technology
Link
http://www.nature.com/articles/nri1804.pdf
Reference119 articles.
1. Savitsky, K. et al. A single ataxia telangiectasia gene with a product similar to PI-3 kinase. Science 268, 1749–1753 (1995).The discovery of the protein kinase ATM is a milestone in the studies of cellular responses to DNA DSB damage.
2. Shiloh, Y. ATM and related protein kinases: safeguarding genome integrity. Nature Rev. Cancer 3, 155–168 (2003).
3. Bakkenist, C. J. & Kastan, M. B. DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation. Nature 421, 499–506 (2003).This paper describes that ATM is activated through autophosphorylation.
4. Assenmacher, N. & Hopfner, K. P. MRE11/RAD50/NBS1: complex activities. Chromosoma 113, 157–166 (2004).
5. Celeste, A. et al. Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks. Nature Cell Biol. 5, 675–679 (2003).
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