MAPKAP Kinase 2 (MK2)-Dependent and -Independent Models of Blister Formation in Pemphigus Vulgaris
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference32 articles.
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2. Autoantibodies against the amino-terminal cadherin-like binding domain of pemphigus vulgaris antigen are pathogenic;Amagai;J Clin Invest,1992
3. Induction of p38MAPK and HSP27 phosphorylation in pemphigus patient skin;Berkowitz;J Invest Dermatol,2007
4. Desmosome signaling. Inhibition of p38MAPK prevents pemphigus vulgaris IgG-induced cytoskeleton reorganization;Berkowitz;J Biol Chem,2005
5. p38MAPK inhibition prevents disease in pemphigus vulgaris mice;Berkowitz;Proc Natl Acad Sci USA,2006
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