Targeted colonic claudin-2 expression renders resistance to epithelial injury, induces immune suppression, and protects from colitis-Reference-Cited by-同舟云学术

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Targeted colonic claudin-2 expression renders resistance to epithelial injury, induces immune suppression, and protects from colitis

Published:2014-03-26 Issue:6 Volume:7 Page:1340-1353
ISSN:1933-0219
Container-title:Mucosal Immunology
language:en
Short-container-title:Mucosal Immunol

Author:

Ahmad R,Chaturvedi R,Olivares-Villagómez D,Habib T,Asim M,Shivesh P,Polk D B,Wilson K T,Washington M K,Van Kaer L,Dhawan P,Singh A B

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/mi201421.pdf

Reference47 articles.

1. Hollander, D. Intestinal permeability, leaky gut, and intestinal disorders. Curr. Gastroenterol. Rep. 1, 410–416 (1999).

2. Arrieta, M.C., Madsen, K, Doyle, J & Meddings, J. Reducing small intestinal permeability attenuates colitis in the IL10 gene-deficient mouse. Gut 58, 41–48 (2009).

3. Tsukita, S & Furuse, M. Occludin and claudins in tight-junction strands: leading or supporting players? Trends Cell Biol. 9, 268–273 (1999).

4. Heller, F. et al. Interleukin-13 is the key effector Th2 cytokine in ulcerative colitis that affects epithelial tight junctions, apoptosis, and cell restitution. Gastroenterology 129, 550–564 (2005).

5. Zeissig, S. et al. Changes in expression and distribution of claudin 2, 5 and 8 lead to discontinuous tight junctions and barrier dysfunction in active Crohn's disease. Gut 56, 61–72 (2007).

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