N-n-Butyl haloperidol iodide ameliorates liver fibrosis and hepatic stellate cell activation in mice-Reference-Cited by-同舟云学术

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N-n-Butyl haloperidol iodide ameliorates liver fibrosis and hepatic stellate cell activation in mice

Published:2021-03-23 Issue:1 Volume:43 Page:133-145
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Shen Dai-fei,Cheng He,Cai Bo-zhi,Cai Wen-feng,Wang Bin,Zhu Qing,Wu Yue-bin,Liu Man,Chen Run-ji,Gao Fen-fei,Zhang Yan-mei,Niu Yong-dong,Shi Gang-gang

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

https://www.nature.com/articles/s41401-021-00630-7.pdf

Reference49 articles.

1. Lee UE, Friedman SL. Mechanisms of hepatic fibrogenesis. Best Pract Res Clin Gastroenterol. 2011;25:195–206.

2. Bataller R, Brenner DA. Liver fibrosis. J Clin Invest. 2005;115:209–18.

3. Tsuchida T, Friedman SL. Mechanisms of hepatic stellate cell activation. Nat Rev Gastroenterol Hepatol. 2017;14:397–411.

4. Lee YA, Wallace MC, Friedman SL. Pathobiology of liver fibrosis: a translational success story. Gut. 2015;64:830–41.

5. Rotman Y, Sanyal AJ. Current and upcoming pharmacotherapy for non-alcoholic fatty liver disease. Gut. 2017;66:180–90.

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1. Investigating the cardiotoxicity of N-n-butyl haloperidol iodide: Inhibition mechanisms on hERG channels;Toxicology;2024-11

2. Activating Protein-1 (AP-1): A Promising Target for the Treatment of Fibrotic Diseases;Current Medicinal Chemistry;2024-02

3. Aloin Attenuates Oxidative Stress, Inflammation, and CCl₄-Induced Liver Fibrosis in Mice: Possible Role of TGF-β/Smad Signaling;Journal of Agricultural and Food Chemistry;2023-12-01

4. N‐n‐butyl haloperidol iodide mediates cardioprotection via regulating AMPK/FoxO1 signalling;Journal of Cellular and Molecular Medicine;2023-11-21

5. Liver Fibrosis Resolution: From Molecular Mechanisms to Therapeutic Opportunities;International Journal of Molecular Sciences;2023-06-02

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