Targeted inhibition of calpain in mitochondria alleviates oxidative stress-induced myocardial injury-Reference-Cited by-同舟云学术

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Targeted inhibition of calpain in mitochondria alleviates oxidative stress-induced myocardial injury

Published:2020-09-23 Issue:6 Volume:42 Page:909-920
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Zheng Dong,Cao Ting,Zhang Lu-lu,Fan Guo-chang,Qiu Jun,Peng Tian-qing

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

https://www.nature.com/articles/s41401-020-00526-y.pdf

Reference52 articles.

1. Goll DE, Thompson VF, Li H, Wei W, Cong J. The Calpain system. Physiol Rev. 2003;83:731–801.

2. Chen Q, Thompson J, Hu Y, Lesnefsky EJ. Cardiomyocyte specific deletion of p53 decreases cell injury during ischemia-reperfusion: role of mitochondria. Free Radic Biol Med. 2020;158:162–70.

3. Chen K, He L, Li Y, Li X, Qiu C, Pei H, et al. Inhibition of GPR35 preserves mitochondrial function after myocardial infarction by targeting calpain 1/2. J Cardiovasc Pharmacol. 2020;75:556–63.

4. Penela P, Inserte J, Ramos P, Rodriguez-Sinovas A, Garcia-Dorado D, Mayor F Jr. Degradation of GRK2 and AKT is an early and detrimental event in myocardial ischemia/reperfusion. EBioMedicine. 2019;48:605–18.

5. Chen Q, Thompson J, Hu Y, Dean J, Lesnefsky EJ. Inhibition of the ubiquitous calpains protects complex I activity and enables improved mitophagy in the heart following ischemia-reperfusion. Am J Physiol Cell Physiol. 2019;317:C910–C21.

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