Interplay of histidine residues of the Alzheimer’s disease Aβ peptide governs its Zn-induced oligomerization-Reference-Cited by-同舟云学术

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Interplay of histidine residues of the Alzheimer’s disease Aβ peptide governs its Zn-induced oligomerization

Published:2016-02-22 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Istrate Andrey N.,Kozin Sergey A.,Zhokhov Sergey S.,Mantsyzov Alexey B.,Kechko Olga I.,Pastore Annalisa,Makarov Alexander A.,Polshakov Vladimir I.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep21734.pdf

Reference80 articles.

1. Hardy, J. & Selkoe, D. J. The amyloid hypothesis of Alzheimer’s disease: progress and problems on the road to therapeutics. Science 297, 353–356, 10.1126/science.1072994 (2002).

2. Musiek, E. S. & Holtzman, D. M. Three dimensions of the amyloid hypothesis: time, space and ‘wingmen’. Nat Neurosci 18, 800–806, 10.1038/nn.4018 (2015).

3. Meyer-Luehmann, M. et al. Exogenous induction of cerebral beta-amyloidogenesis is governed by agent and host. Science 313, 1781–1784, 10.1126/science.1131864 (2006).

4. Ayton, S., Lei, P. & Bush, A. Biometals and Their Therapeutic Implications in Alzheimer’s Disease. Neurotherapeutics 12, 109–120, 10.1007/s13311-014-0312-z (2015).

5. Ballard, C. et al. Alzheimer’s disease. Lancet 377, 1019–1031, 10.1016/S0140-6736(10)61349-9 (2011).

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