Deficiency in interferon type 1 receptor improves definitive erythropoiesis in Klf1 null mice-Reference-Cited by-同舟云学术

Deficiency in interferon type 1 receptor improves definitive erythropoiesis in Klf1 null mice

Published:2017-12-11 Issue:3 Volume:25 Page:589-599
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Manchinu Maria Francesca,Brancia Carla,Caria Cristian Antonio,Musu Ester,Porcu Susanna,Simbula Michela,Asunis Isadora,Perseu Lucia,Ristaldi Maria Serafina

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/s41418-017-0003-5.pdf

Reference41 articles.

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2. Perkins AC, Sharpe AH, Orkin SH. Lethal beta-thalassaemia in mice lacking the erythroid CACCC-transcription factor EKLF. Nature 1995;375:318–22.

3. Magor GW, et al. KLF1-null neonates display hydrops fetalis and a deranged erythroid transcriptome. Blood 2015;125(15):2405–17.

4. Tallack MR, Perkins AC. KLF1 directly coordinates almost all aspects of terminal erythroid differentiation. IUBMB Life 2010;62(12):886–90.

5. Siatecka M, Bieker JJ. The multifunctional role of EKLF/KLF1 during erythropoiesis. Blood 2011;118:2044–54.

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