New insights into the mechanisms of podocyte health
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
http://www.nature.com/articles/nrneph.2015.204.pdf
Reference10 articles.
1. Dong, L. et al. Integration of cistromic and transcriptomic analyses identifies Nphs2, Mafb, and Magi2 as Wilms' tumor 1 target genes in podocyte differentiation and maintenance. J. Am. Soc. Nephrol. 26, 2118–2128 (2015).
2. Kann, M. et al. Genome-wide analysis of Wilms' tumor 1-controlled gene expression in podocytes reveals key regulatory mechanisms. J. Am. Soc. Nephrol. 26, 2097–2104 (2015).
3. Lefebvre, J. et al. Alternatively spliced isoforms of WT1 control podocyte-specific gene expression. Kidney Int. 88, 321–331 (2015).
4. Ashraf, S. et al. ADCK4 mutations promote steroid-resistant nephrotic syndrome through CoQ10 biosynthesis disruption. J. Clin. Invest. 123, 5179–5189 (2013).
5. Casalena, G. et al. Mpv17 in mitochondria protects podocytes against mitochondrial dysfunction and apoptosis in vivo and in vitro. Am. J. Physiol. Renal Physiol. 306, F1372–F1380 (2014).
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