Angiotensin-(1–7) abrogates angiotensin II-induced proliferation, migration and inflammation in VSMCs through inactivation of ROS-mediated PI3K/Akt and MAPK/ERK signaling pathways-Reference-Cited by-同舟云学术

Angiotensin-(1–7) abrogates angiotensin II-induced proliferation, migration and inflammation in VSMCs through inactivation of ROS-mediated PI3K/Akt and MAPK/ERK signaling pathways

Published:2016-09-30 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Zhang Feng,Ren Xingsheng,Zhao Mingxia,Zhou Bing,Han Ying

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep34621.pdf

Reference56 articles.

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3. Ragolia, L., Palaia, T., Paric, E. & Maesaka, J. K. Prostaglandin D2 synthase inhibits the exaggerated growth phenotype of spontaneously hypertensive rat vascular smooth muscle cells. J Biol Chem 278, 22175–22181 (2003).

4. Rudijanto, A. The role of vascular smooth muscle cells on the pathogenesis of atherosclerosis. Acta Med Indones 39, 86–93 (2007).

5. ROSS, R. Atherosclerosis–an inflammatory disease. N Engl J Med. 340, 115–126 (1999).

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