Glutamatergic synaptic input to glioma cells drives brain tumour progression-Reference-Cited by-同舟云学术

Glutamatergic synaptic input to glioma cells drives brain tumour progression

Published:2019-09-18 Issue:7775 Volume:573 Page:532-538
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Venkataramani Varun,Tanev Dimitar Ivanov,Strahle Christopher,Studier-Fischer Alexander,Fankhauser Laura,Kessler Tobias,Körber Christoph,Kardorff Markus,Ratliff Miriam,Xie Ruifan,Horstmann Heinz,Messer Mirko,Paik Sang Peter,Knabbe Johannes,Sahm Felix,Kurz Felix T.,Acikgöz Azer Aylin,Herrmannsdörfer Frank,Agarwal Amit,Bergles Dwight E.,Chalmers Anthony,Miletic Hrvoje,Turcan Sevin,Mawrin Christian,Hänggi Daniel,Liu Hai-Kun,Wick Wolfgang,Winkler Frank,Kuner Thomas

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-019-1564-x.pdf

Reference37 articles.

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2. Venkatesh, H. S. et al. Neuronal activity promotes glioma growth through neuroligin-3 secretion. Cell 161, 803–816 (2015).

3. Venkatesh, H. S. et al. Targeting neuronal activity-regulated neuroligin-3 dependency in high-grade glioma. Nature 549, 533–537 (2017).

4. Ishiuchi, S. et al. Blockage of Ca2+-permeable AMPA receptors suppresses migration and induces apoptosis in human glioblastoma cells. Nat. Med. 8, 971–978 (2002).

5. Takano, T. et al. Glutamate release promotes growth of malignant gliomas. Nat. Med. 7, 1010–1015 (2001).

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