Excess of NPM-ALK oncogenic signaling promotes cellular apoptosis and drug dependency-Reference-Cited by-同舟云学术

Excess of NPM-ALK oncogenic signaling promotes cellular apoptosis and drug dependency

Published:2015-12-14 Issue:29 Volume:35 Page:3854-3865
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Ceccon M,Merlo M E Boggio,Mologni L,Poggio T,Varesio L M,Menotti M,Bombelli S,Rigolio R,Manazza A D,Di Giacomo F,Ambrogio C,Giudici G,Casati C,Mastini C,Compagno M,Turner S D,Gambacorti-Passerini C,Chiarle R,Voena C

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2015456.pdf

Reference55 articles.

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3. Morris SW, Kirstein MN, Valentine MB, Dittmer KG, Shapiro DN, Saltman DL et al. Fusion of a kinase gene, ALK, to a nucleolar protein gene, NPM, in non-Hodgkin's lymphoma. Science 1994; 263: 1281–1284.

4. Pulford K, Morris SW, Turturro F . Anaplastic lymphoma kinase proteins in growth control and cancer. J Cell Physiol 2004; 199: 330–358.

5. Bischof D, Pulford K, Mason DY, Morris SW . Role of the nucleophosmin (NPM) portion of the non-Hodgkin's lymphoma-associated NPM-anaplastic lymphoma kinase fusion protein in oncogenesis. Mol Cell Biol 1997; 17: 2312–2325.

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