FcRγ-dependent immune activation initiates astrogliosis during the asymptomatic phase of Sandhoff disease model mice-Reference-Cited by-同舟云学术

FcRγ-dependent immune activation initiates astrogliosis during the asymptomatic phase of Sandhoff disease model mice

Published:2017-01-13 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Ogawa Yasuhiro,Sano Takafumi,Irisa Masahiro,Kodama Takashi,Saito Takahiro,Furusawa Eiri,Kaizu Katsutoshi,Yanagi Yusuke,Tsukimura Takahiro,Togawa Tadayasu,Yamanaka Shoji,Itoh Kohji,Sakuraba Hitoshi,Oishi Kazuhiko

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep40518.pdf

Reference52 articles.

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2. Sango, K. et al. Mouse models of Tay-Sachs and Sandhoff diseases differ in neurologic phenotype and ganglioside metabolism. Nature genetics 11, 170–176, doi: 10.1038/ng1095-170 (1995).

3. Phaneuf, D. et al. Dramatically different phenotypes in mouse models of human Tay-Sachs and Sandhoff diseases. Human molecular genetics 5, 1–14 (1996).

4. Sango, K. et al. Mice lacking both subunits of lysosomal beta-hexosaminidase display gangliosidosis and mucopolysaccharidosis. Nature genetics 14, 348–352, doi: 10.1038/ng1196-348 (1996).

5. Volk, B. W. Pathologic anatomy 36–67 (Grune and Stratton, Inc., 1964).

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