DMP1 prevents osteocyte alterations, FGF23 elevation and left ventricular hypertrophy in mice with chronic kidney disease-Reference-Cited by-同舟云学术

DMP1 prevents osteocyte alterations, FGF23 elevation and left ventricular hypertrophy in mice with chronic kidney disease

Published:2019-04-25 Issue:1 Volume:7 Page:
ISSN:2095-6231
Container-title:Bone Research
language:en
Short-container-title:Bone Res

Author:

Dussold Corey,Gerber Claire,White Samantha,Wang Xueyan,Qi Lixin,Francis Connor,Capella Maralee,Courbon Guillaume,Wang Jingya,Li Chaoyuan,Feng Jian Q.,Isakova Tamara,Wolf Myles,David Valentin,Martin Aline

Funder

U.S. Department of Health & Human Services | NIH | National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Springer Science and Business Media LLC

Subject

Physiology,Histology,Endocrinology, Diabetes and Metabolism

Link

http://www.nature.com/articles/s41413-019-0051-1.pdf

Reference46 articles.

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2. Malluche, H. H., Mawad, H. & Monier-Faugere, M. C. The importance of bone health in end-stage renal disease: out of the frying pan, into the fire? Nephrol. Dial. Transplant. 19(Suppl 1), i9–i13 (2004).

3. Shimada, T. et al. Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism. J. Clin. Invest. 113, 561–568 (2004).

4. Shimada, T. et al. FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis. J. Bone Miner. Res. 19, 429–435 (2004).

5. Isakova, T. et al. Fibroblast growth factor 23 is elevated before parathyroid hormone and phosphate in chronic kidney disease. Kidney Int. 79, 1370–1378 (2011).

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