Inhibition of endogenous SPARC enhances pancreatic cancer cell growth: modulation by FGFR1-III isoform expression
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology
Link
http://www.nature.com/articles/6605440.pdf
Reference22 articles.
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2. Bradshaw AD, Sage EH (2001) SPARC, a matricellular protein that functions in cellular differentiation and tissue response to injury. J Clin Invest 107: 1049–1054
3. Cheetham S, Tang MJ, Mesak F, Kennecke H, Owen D, Tai IT (2008) SPARC promoter hypermethylation in colorectal cancers can be reversed by 5-Aza-2′deoxycytidine to increase SPARC expression and improve therapy response. Br J Cancer 98 (11): 1810–1819
4. Chen G, Wang J, Liu Z, Kornmann M (2008) Exon III splicing of fibroblast growth factor receptor 1 is modulated by growth factors and cyclin D1. Pancreas 37: 159–164
5. Clark CJ, Sage EH (2008) A prototypic matricellular protein in the tumor microenvironment – where there's SPARC, there's fire. J Cell Biochem 104 (3): 721–732
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