A potassium channel mutation in weaver mice implicates membrane excitability in granule cell differentiation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics
Link
http://www.nature.com/articles/ng1095-126.pdf
Reference37 articles.
1. Lane, P.W. Mouse News Lett. 30–32 (1964).
2. Rakic, P. & Sidman, R.L. Sequence of developmental abnormalities leading to granule cell deficit in cerebellar cortex of weaver mutant mice. J. comp. Neurol. 152, 103–132 (1973).
3. Rakic, P. & Sidman, R.L. Organization of cerebellar cortex secondary to deficit of granule cells in weaver mutant mice. J. Comp. Neurol. 152, 133–161 (1973).
4. Rakic, P. & Sidman, R.L. Weaver mutant mouse cerebellum: defective neuroal migration secondary to abnormality of Bergmann glia. Proc. natn. Acad. Sci. U.S.A. 70, 240–244 (1973).
5. Rezai, Z. & Yoon, C.H. Abnormal rate of granule cell migration in the cerebellum of “weaverrdquo; mutant mice. Dev. Biol. 29, 17–26 (1972).
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