A recombination–based assay demonstrates that the fragile X sequence is transcribed widely during development
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics
Link
http://www.nature.com/articles/ng0193-44.pdf
Reference25 articles.
1. Watt, V.M., Ingles, C.J., Urdea, M.S. & Rutter, W.J. Homology requirements for recombination in Escherichia coli. Proc. natn. Acad. Sci. U.S.A. 82, 4768–4772 (1985).
2. Shen, P. & Huang, H.V. Homologous recombination in Escherichia coli: dependence on substrate length and homology. Genetics 112, 441–457 (1986).
3. King, S.R. & Richardson, J.P. Role of homology and pathway specificity for recombination between plasmids and bacteriophage λ. Molec. gen. Genet. 204, 141–147, (1986).
4. Shen, P. & Huang, H.V. Effect of base pair mismatches on recombination via the RecBCD pathway. Molec. gen. Genet. 218, 358–360 (1989).
5. Verkerk, A.J. et al. Identification of a gene (FMR-1) containing a CGG repeat coincident with a breakpoint cluster region exhibiting length variation in fragile X syndrome. Cell 65, 905–914 (1991).
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