A framework for interpreting genome-wide association studies of psychiatric disorders

Author:

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Reference28 articles.

1. Pe’er I, Yelensky R, Altshuler D, Daly MJ . Estimation of the multiple testing burden for genomewide association studies of nearly all common variants. Genet Epidemiol 2008; 32: 381–385.

2. Weiss LA, Shen Y, Korn JM, Arking DE, Miller DT, Fossdal R et al. Association between microdeletion and microduplication at 16p11.2 and autism. N Engl J Med 2008; 358: 667–675.

3. Ferreira M, O’Donovan M, Meng Y, Jones I, Ruderfer D, Jones L et al. Collaborative genome-wide association analysis of 10,596 individuals supports a role for Ankyrin-G (ANK3) and the alpha-1C subunit of the L-type voltage-gated calcium channel (CACNA1C) in bipolar disorder. Nat Genet 2008; e-pub ahead of print 2008 August 17.

4. O’Donovan M, Craddock N, Norton N, Williams H, Peirce T, Moskvina V et al. Identification of novel schizophrenia loci by genome-wide association and follow-up. Nat Genet 2008; e-pub ahead of print 2008 July 30.

5. International Schizophrenia Consortium. Greater burden of rare copy number variants in schizophrenia. Nature 2008; 455: 237–241.

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