Gingival fibroblasts resist apoptosis in response to oxidative stress in a model of periodontal diseases-Reference-Cited by-同舟云学术

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Gingival fibroblasts resist apoptosis in response to oxidative stress in a model of periodontal diseases

Published:2015-11-09 Issue:1 Volume:1 Page:
ISSN:2058-7716
Container-title:Cell Death Discovery
language:en
Short-container-title:Cell Death Discovery

Author:

Cheng R,Choudhury D,Liu C,Billet S,Hu T,Bhowmick NA

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddiscovery201546.pdf

Reference47 articles.

1. Page RC, Offenbacher S, Schroeder HE, Seymour GJ, Kornman KS . Advances in the pathogenesis of periodontitis: summary of developments, clinical implications and future directions. Periodontol 2000 1997; 14: 216–248.

2. Gorąca A, Huk-Kolega H, Kleniewska P, Piechota-Polańczyk A, Skibska B . Effects of lipoic acid on spleen oxidative stress after LPS administration. Pharmacol Rep 2013; 65: 179–186.

3. Melo ES, Barbeiro HV, Ariga S, Goloubkova T, Curi R, Valasco IT et al. Immune cells and oxidative stress in the endotoxin tolerance mouse model. Braz J Med Biol Res 2010; 43: 57–67.

4. Sanikidze TV, Tkhilava NG, Papava MB, Datunashvili IV, Gongadze MT, Gamrekelashvili DD et al. Role of free nitrogen and oxygen radical in the pathogenesis of lipopolysaccharide-induced endotoxemia. Bull Exp Biol Med 2006; 141: 211–215.

5. Bykov I, Ylipaasto P, Erola L, Lindros KO . Phagocytosis and LPS stimulated production of cytokines and prostaglandin E2 is different in Kupffer cells isolated from the periportal or perivenous liver region. Scand J Gastroenterol 2003; 38: 1256–1261.

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